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Critical role of oxidative stress and sustained JNK activation in aloe-emodin-mediated apoptotic cell death in human hepatoma cells

This review summarizes the roles of ferroptosis and mitochondrial injury in the progression of DCM and explores the molecular mechanisms involved, with an emphasis on the interplay between these two processes

This cytokine modulation appears to be a key mechanism underlying its efficacy in models of inflammatory bowel disease, surgical wound healing, and systemic inflammatory injury

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